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In the case of humoral immunity, B cells are activated by interaction with T cells through the antigen presentation via the B cell receptor on the B cell surface. Activated B cells then proliferate and differentiate into memory cells or plasma cells that produce immunoglobulins (Ig). These antibodies may neutralize the pathogen, thereby preventing the spread of the infection.
However, upon being activated by the antigen, certain T cells may secrete cytokines that result in the production of lymphokines by neighboring cells. Some of these lymphokines cause an increase in the proliferation and differentiation of cells within the T cell population. These cytokines are normally only produced by activated T cells, but can be produced by a T cell that has encountered an antigen (antigen-specific T cell). This phenomenon is known as T cell clonal expansion, which results in an increase of cells (e.g., T cells) that share a common T cell receptor (TCR). For example, a T cell that is activated by antigen presented by class II major histocompatibility complex (MHC) molecules can stimulate its neighbors via the secretion of a lymphokine called interleukin-2 (IL-2). Because IL-2 is required for the growth of T cells, the production of IL-2 by 0b46394aab
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